akroeze
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Posts posted by akroeze
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Her sats were dropping but by no means poor. She had gone from 99% at onset to 93% when we were there. A relative drop of 6% but still not a worrying number on its own.Just curious what your rationale is for not giving adrenaline. You said she has poor sats,
any other ss/sx worth mentioning? pulse and BP? resp. rate? chest sounds? work of breathing? general appearance? rash? nausea/vomiting? altered conscious state? etc etcAll V/S are appropriate with clear air entry and patient is resting comfortably.
I guess what I'm getting at is what else are you waiting for? and what are your indications for giving adrenaline?Anaphylaxis, which this was definitely not. Although if we got into airway compromise you could argue that it is in that realm.
That does bring up an interesting point... is angioedema from ACEi REALLY an allergic reaction? From the reading I have been doing it seems there is no consensus. The general impression I get though is that it isn't really as histamine is not involved. The "best guess" is that ACEi also inhibit the breakdown of bradykinin which builds up and causes the angioedema. So is this truly an allergic reaction? Do they fall into a medical directive that is for "allergic reactions"?
The pt got 50mg PO of benadryl, remember all drugs thet go enteral are subject to first pass metabolism. The dose was also 3hrs ago. so really, as far as bloodserum levels are concerned, she is not "maxed out" as far as therapeutic index goes. In fact, she is probably below.
I would give 50IV myself.
I can see that argument.
I would have no problem trialing a local epi via nebulizer to vasoconstrict and somewhat make her more comfortable.In all honesty the thought didn't even occur to me until post-call (and of course I would have to receive an order from my base hospital physician to do it).
Also, getting a little epi into the system is always a good thing during allergic reactions with airway comprimise as it does in fact stabilize the MAST cell, therefore inhibiting the release of more histamine.Is Epi going to help angioedema? Epi helps edema in anaphylaxis (as far as I understand it) by "reversing" the fluid leakage into the interstitial tissue... is the same pathology present in angioedema? I honestly don't know.
I also may hit her with a Dexamethosone, or prednisone. It will help with the swelling somewhat, but moreso, we will be one step ahead in case this does go for shit later.Yeah, totally agree there. Unfortunately steroids are not in my arsenal. Otherwise it is probably the most ideal option.
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Recently had an elderly female patient from a nursing home who had developed tongue swelling in the am which was progressively worsening. She is on Coversyl. 3hrs prior to EMS the staff administered 50mg Diphenhydramine PO with no noticeable effect. EMS was summoned when patient's sats were starting to drop and she was having a hard time speaking due to the edema.
Are there any pre-hospital treatments that are effective here? She has already had a therapeutic dose of Benadryl therefore giving more is probably not indicated. She is not in extremis (and is 97 and has a LONG cardiac history) therefore Epi is probably not a great choice at this time, at least IM. Is there any benefit to nebulized Epi here? What other medications may help this patient?
Cheers
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I'm not sure if I'm going to go or not.... I am saving up for a down payment on a house and now live at the other end of the province so travel there would be more costly. We'll see.
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Hey all, just thought I'd link you to a great site with lots of broadcasts about rhythm and 12 lead interpretation.
These casts were done by my ACP instructor and a VERY well known figure in Ontario (and elsewhere) EMS
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To me it really is one that if they are stable then the best option may be to just monitor them and be conservative with treatment. An expert really may be the best choice for these patients.
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My bad for attempting to share what I know. I now know that next time I want to offer help here I'll just shut up, read, and snicker to myself.
Wow, sensitive and over react much?
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To satisfy your curiosity - I am a fairly new medic with 1 year of experience. I run in a very high volume environment and in the year, I have about 1200 calls under my belt. I am scrambling as fast as I can to become a good street medic. The challenges of this position leave me exhausted. I personally run an AVERAGE of 20 calls in a 48 hour shift. Standup 48's (where we get little to no sleep at all) are not rare. I figure another year on the street, then whammo - more education.
It just blows me away that that is legal. My usual 12hr shift is non-stop run run run go go go and I am exhausted at the end of it. I can't imagine effectively and safely doing it 4 times in a row.
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Spenac, I don't know if I advocate a 15-lead on every patient. There are indications, which can be found on a 12-lead, for 15-leads.
EMS_Cadet
Where have you read that Mobitz 2 almost always originates in the bundle branches? Mobitz 2 is commonly an infranodal block, but of the his bundle above the bundle branches. .
Click >>>THE ANSWER
Your link is broken!
Here is a working one:
Click >>>THE ANSWER
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Surely there isn't any dye in the intravenous formulation?
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My interpretation would be an untypable 2nd degree AVB
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Has anyone heard of someone being allergic to Diphenhydramine?
I had an allergic reaction case yesterday that became anaphylactic immediately after I administered IV Benadryl. I know the far more likely case is that that is coincidence and the patient just chose that moment to go anaphylactic from her bee sting, but just wondering if it is possible?
Full details here:
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On a somewhat related note, the other night I did my first intubation as an independently practicing ACP
Got it on the first try in a cramped room where I was positioned more beside the patient than at the top of their head.
Sorry, just wanted to congratulate myself
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JUST TAKE THE EMT COURSE IN THE LONG RUN YOULL BE HAPPY YOU TOOK IT YOULL NEVER KNOW WHEN IT CAN SAVE YOU TIME
WHY ARE YOU YELLING?
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Well looks like in Ontario we are right in the middle of the pack as far as that goes.
When the patient is symptomatically hypotensive/hypovolemic without signs of fluid overload on chest auscultation, and has a systolic BP<100 (or SBP<[2 x patient age + 70] in patient <40kg) the paramedic may:a. For patients >=40kg: Give an IV fluid bolus to a maximum of 20ml/kg. Repeat vitals and perform a chest auscultations after every 250cc. Return to KVO when bolus completed, SBP is >=100 or chest auscultation reveals crackles.
b. For patients <40kg: Give an IV fluid bolus to a maximum of 20ml/kg. Repeat vitals and perform a chest auscultations after every 100cc. Return to KVO when bolus completed, SBP is >=(2 x patient age + 70) or chest auscultation reveals crackles. In patients <40kg with suspected diabetic ketoacidosis, give IV fluid boluses to a maximum of 10ml/kg.
So basically we are trying to keep them at 100 systolic.
Interestingly our post-arrest protocol calls for maintaining BP >90 systolic.
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Speaking both flippantly, and off the top of my head, EMT-Bs transport patients, FRs don't.
Yes but I'm asking what the practical difference is other than that. Is an EMT-B just a FR who can drive the patient somewhere?
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Good on him. Although I do have to say he shouldn't have jumped out of the moving vehicle like that.
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I know for sure EMT-Bs can administer oxygen, place oral and nasal airways, as well as combitubes. Not sure what else.
First responders can't?
Here the 40hr FR course gets you:
O2, Defib, OPA, NPA, Backboarding, KED, Traction Splint and Vital Signs (including BP) training
Only thing they don't get that you mentioned is combitube
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What can an EMT-B do that a FR can't? Just curious.
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It works, I've done it. Although, I found that more force needed to be applied than the prescribed "let your fist drop onto pt's chest," making it more of a precordial thump, but aimed just left of the lower end of the sternum. I'd say I used moderate force with my hand 6-8" above pt chest, and it definitely produced perfusing pacing beats.
Attached is a snippet of the strip. The difference in beats, seemed to be related to the force used.
With percussion pacing, though, I imagine you would have to worry about R on T phenomenon, or commotio cordis...
Do your protocols allow for it?
What made you decide to do this rather than putting the pads on and electrically pacing?
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I use the scoop all the time. I prefer it to the LSB. Also any suspected hip or pelvic fx is getting it over the long board.
Ditto, I love the scoop although there are times when an LSB is more practical (vehicle extrication for example)
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Ok, I gotta ask. Why did you post this thread if you aren't saying anything? Your post was basically pointless. Until you have proof then say nothing...
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It is common practice here that if one gets a call for a priority transfer out of town very close to the end of shift then they will go about it as usual but if the oncoming crew gets to base in time they will bring the spare truck over and swap out with the offgoing crew. Never is transport delayed in order to facilitate this.
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We had this patient last week who presented with dizziness and loss of balance when standing. He also began complaining of pain in his upper and lower extremities and all 4 Qs of his abdomen. En route he also began experiencing CP. He had no history of MIs or CPs prior, his EKG and VS were all normal. But he did have lung cancer and was undergoing hemotherapy which he had just finished the previous week. Could that be the cause of all his symptomps?
Short answer is yes, it could be the answer. One of many many many possible answers.
ACEi Angioedema
in Patient Care
Posted
Unfortunately in Ontario we do not do RSI nor do we do facilitated intubation (it is EXTREMELY frowned upon at least), and at least in my particular region Cric is no longer in my tool box. So essentially I have routine intubation with no sedation and topical lidocaine only as an option or blind nasotracheal.