We have had some interesting results since we swapped to Ketamine as our primary induction agent with our status patients. Where previously they were unresponsive to Midazolam, they often cease seizing on induction. As we do not routinely paralyse our status patients unless their seizure activity prevents their oxygenation, so the tube is maintained with sedation alone.
Previously when our induction was Fentanyl/Midazolam, you would get brief periods where the patient would cease seizing but then recommence and you were often bolusing midazolam during transport on top of you sedation.
I love Ketamine so much, can't remember life before ketamine (I don't want to remember it either)